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Ceprandal (Omeprazole)

Ceprandal target is the treatment of symptoms of gastroesophageal reflux disease (GERD) and other conditions caused by excess stomach acid. It is also taken to promote healing of erosive esophagitis (damage to your esophagus caused by stomach acid). Ceprandal is acting by decreasing the amount of acid produced in the stomach. It is proton pump inhibitor (PPI). Ceprandal is available in tablets (10 mg, 20 mg, 40 mg) and capsules.

Other names for this medication:
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Ceprandal is used to treat certain conditions where there is too much acid in the stomach. It is used to treat gastric and duodenal ulcers, erosive esophagitis, and gastroesophageal reflux disease (GERD). GERD is a condition where the acid in the stomach washes back up into the esophagus. Sometimes Ceprandal is used in combination with antibiotics (eg, amoxicillin, clarithromycin) to treat ulcers associated with the infection caused by the H. pylori bacteria.

Ceprandal is also used to treat Zollinger-Ellison syndrome, a condition where the stomach produces too much acid.

Ceprandal is also used to treat dyspepsia, a condition that causes sour stomach, belching, heart burn, or indigestion.

In addition, Ceprandal is used to prevent upper gastrointestinal tract bleeding in seriously ill patients.

Ceprandal is a proton pump inhibitor (PPI). It works by decreasing the amount of acid produced by the stomach.

This medicine is available both over-the-counter (OTC) and with your doctor's prescription.

This product is available in the following dosage forms:

  • Powder for Suspension
  • Tablet Disintegrating, Delayed Release
  • Capsule, Delayed Release
  • Tablet, Delayed Release
  • Packet

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Using automation to control receivables: how computers can improve the revenue cycle. Effectiveness of detection and stimulation of myocardium with titanium electrodes coated with titanium nitride Altered Daytime Fluctuation Pattern of Plasminogen Activator Inhibitor 1 in Type 2 Diabetes Patients with Coronary Artery Disease: A Strong Association with Persistently Elevated Plasma Insulin, Increased Insulin Resistance, and Abdominal Obesity. After eight weeks of corticosteroid therapy, the bronchoalveolar lavage eosinophil population and the technetium-99m glucoheptonate uptake had returned to normal.

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